When Jodi Ferris unknowingly unleashed the wrath of Social Services at Penn State Hershey Medical Center by questioning the necessity of the Hepatitis B vaccine for her newborn daughter, little did she know just how big a can of worms she was opening.
A study reported in the journal Apoptosis just weeks ago indicates that this controversial vaccine normally injected into newborns within hours of birth induces liver damage primarily due to the presence of the toxic vaccine adjuvant aluminum hydroxide.
Aluminum hydroxide, an adjuvant also present in the anthrax vaccine, has already been shown as a likely culprit for triggering the motor neuron autoimmunity issues present in Gulf War Syndrome which are indistinguishable from the autoimmune disease amyotrophic lateral sclerosis (ALS).
Vaccine adjuvants like aluminum hydroxide are the dirty little secret that the scientific community doesn’t want the public to know about. Their purpose is to stimulate and heighten the immune response to the vaccine itself.
In this new study, aluminum hydroxide induced loss of mitochondrial integrity and cell death in mouse liver cells exposed to low doses of the Hepatitis B vaccine containing the toxic adjuvant.
The mitochondria is the “powerhouse” of a cell where energy is created and cellular respiration occurs. It is not surprising that compromising the mitochondria of liver cells results in outright death of the cell itself.
This new study corroborates other research indicating liver damage from Hep B. In 1999, it was found that children less than 6 years old had a nearly 300% increase in the incidence of liver problems if they had been vaccinated with Hepatitis B versus those children that did not have the Hep B jab.
It’s not just the young developing livers of children that suffer from the toxic Hep B. The Journal Hepatogastroenterology reported in 2002 that the Hepatitis B vaccine was statistically associated with increased risk for hepatitis, gastrointestinal disease, and liver function test abnormalities in adult females.
Did you get that?
The Hepatitis B vaccine is statistically associated with increased risk for hepatitis in adults!
A more recent study in June 2011 reported by the journal Molecular Biology Reports indicates that damage from Hep B is likely caused by alteration in the expression of 144 genes in mouse livers within one day of vaccination, 7 of which are associated with inflammation and metabolism.
The Only Sane Answer is Rejection of the Hepatitis B Vaccine
The thing that makes absolutely zero sense is why the controversial and very questionable Hepatitis B vaccine is still being recommended as standard for all newborns within hours of birth especially given that a newborn’s liver does not even begin functioning until several days later.
With no functioning liver to assist with detoxification, the damaging effects of the toxic Hep B ingredients would no doubt be magnified with the risk of irreparable harm to the newborn great.
If you are a parent with a baby on the way, please do your research and learn the extreme danger of the Hepatitis B vaccine to your newborn.
Even if you have been supportive of vaccination in the past, at least skip this one controversial shot that has so much documented literature about its damaging effects.
There is simply no logical reason to vaccinate all newborns against a disease that is primarily sexually transmitted or contracted via the used needles of drug addicts. Better to screen mothers preemptively rather than vaccinate all newborns with this dangerous drug cocktail that threatens their long term health.
Sarah, The Healthy Home Economist
Source: Hepatitis B Vaccine Induces Apoptotic Death in Hepa1-6 Cells, January 12, 2012
The Healthy Home Economist holds a Master’s degree from the University of Pennsylvania. Mother to 3 healthy children, blogger, and best-selling author, she writes about the practical application of Traditional Diet and evidence-based wellness within the modern household. Her work has been featured by USA Today, The New York Times, National Review, ABC, NBC, and many others.